Trypanosomacruzi-Infected Pregnant Women without Vector Exposure Have Higher Parasitemia Levels: Implications for Congenital Transmission Risk

dc.contributor.authorRendell, Victoria R.
dc.contributor.authorGilman, Robert H.
dc.contributor.authorValencia, Edward
dc.contributor.authorGaldos-Cardenas, Gerson
dc.contributor.authorVerastegui, Manuela
dc.contributor.authorSanchez, Leny
dc.contributor.authorAcosta, Janet
dc.contributor.authorSanchez, Gerardo
dc.contributor.authorFerrufino, Lisbeth
dc.contributor.authorLaFuente, Carlos
dc.contributor.authorAbastoflor, Maria del Carmen
dc.contributor.authorColanzi, Rony
dc.contributor.authorBern, Caryn
dc.date.accessioned2022-11-10T02:41:08Z
dc.date.available2022-11-10T02:41:08Z
dc.date.issued2015-01-14
dc.description.abstractCongenital transmission is a major source of new Trypanosoma cruzi infections, and as vector and blood bank control continue to improve, the proportion due to congenital infection will grow. A major unanswered question is why reported transmission rates from T.cruzi-infected mothers vary so widely among study populations. Women with high parasite loads during pregnancy are more likely to transmit to their infants, but the factors that govern maternal parasite load are largely unknown. Better understanding of these factors could enable prioritization of screening programs to target women most at risk of transmission to their infants.es_ES
dc.identifier.urihttps://repositorio.ucb.edu.bo/handle/20.500.12771/554
dc.language.isoenes_ES
dc.subjectTrypanosoma cruzi-Infected Pregnantes_ES
dc.subjectCongenital Transmission Riskes_ES
dc.titleTrypanosomacruzi-Infected Pregnant Women without Vector Exposure Have Higher Parasitemia Levels: Implications for Congenital Transmission Riskes_ES
dc.typeArticlees_ES

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